LIF- and IL-6-Induced Acetylation of STAT3 at Lys-685 through PI3K/Akt Activation

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LIF- and IL-6-induced acetylation of STAT3 at Lys-685 through PI3K/Akt activation.

Signal transducer and activator of transcription 3 (STAT3), which mediates biological actions in many physiological processes, is activated by cytokines and growth factors via specific tyrosine or serine phosphorylation, dimerization and nuclear translocation. A recent study has demonstrated, by using antibody to acetylated lysine, and a STAT3 mutant with Lys-685-to-Arg substitution, that STAT3...

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Biol. Pharm. Bull. 30(10) 1860—1864 (2007)

is a member of the STAT protein family, and is mainly activated by the interleukin (IL)-6 family of cytokines, interferons, epidermal growth factor and leptin. Similar to other members of the STAT family, upon cytokine stimulation, STAT3 is tyrosine-phosphorylated at Tyr-705 by Janus kinases (Jaks), and is subsequently dimerized and translocated to the nucleus to activate its target genes. STAT...

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The IL-6 family of cytokines modulates STAT3 activation by desumoylation of PML through SENP1 induction.

Post-translational modification by small ubiquitin-like modifier (SUMO) plays an important role in the regulation of different signaling pathways and is involved in the formation of promyelocytic leukemia (PML) protein nuclear bodies following sumoylation of PML. In the present study, we found that IL-6 induces desumoylation of PML and dissociation between PML and SUMO1 in hepatoma cells. We al...

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Rac1 mediates STAT3 activation by autocrine IL-6.

The activity of the small GTPase, Rac1, plays a role in various cellular processes including cytoskeletal rearrangement, gene transcription, and malignant transformation. In this report constitutively active Rac1 (Rac V12) is shown to stimulate the activation of STAT3, a member of the family of signal transducers and activators of transcription (STATs). The activity of Rac1 leads to STAT3 trans...

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NF-κB-Induced IL-6 Ensures STAT3 Activation and Tumor Aggressiveness in Glioblastoma

Glioblastoma (GBM) is the most aggressive, neurologically destructive and deadly tumor of the central nervous system (CNS). In GBM, the transcription factors NF-κB and STAT3 are aberrantly activated and associated with tumor cell proliferation, survival, invasion and chemoresistance. In addition, common activators of NF-κB and STAT3, including TNF-α and IL-6, respectively, are abundantly expres...

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ژورنال

عنوان ژورنال: Biological and Pharmaceutical Bulletin

سال: 2007

ISSN: 0918-6158,1347-5215

DOI: 10.1248/bpb.30.1860